Clofarabine

證據等級: L5 預測適應症: 10

目錄

  1. Clofarabine
  2. Clofarabine: From Acute Lymphoblastic Leukemia to Myeloid Leukemia
    1. One-Sentence Summary
    2. Quick Overview
    3. Why is This Prediction Reasonable?
    4. Clinical Trial Evidence
    5. Literature Evidence
    6. Finland Market Information
    7. Cytotoxicity
    8. Safety Considerations
    9. Conclusion and Next Steps
    10. Disclaimer

## 藥師評估報告

Clofarabine: From Acute Lymphoblastic Leukemia to Myeloid Leukemia

One-Sentence Summary

Clofarabine is a second-generation purine nucleoside analog originally developed and FDA-approved for relapsed/refractory acute lymphoblastic leukemia (ALL) in pediatric patients (this approval history is documented within the trial records in this evidence pack, as Taiwan/Finland regulatory licensing data is unavailable). The TxGNN model predicts it may also be effective for Myeloid Leukemia, with 50 clinical trials and 20 publications currently supporting this direction.


Quick Overview

Item Content
Original Indication Relapsed/refractory acute lymphoblastic leukemia (ALL) in pediatric patients (FDA-approved; per clinical trial records — no Finland/Taiwan regulatory license on file)
Predicted New Indication Myeloid Leukemia
TxGNN Prediction Score 99.88%
Evidence Level L2
Finland Market Status 未上市 (Not marketed)
Number of Authorizations 0
Recommended Decision Proceed with Guardrails

Why is This Prediction Reasonable?

Detailed regulatory-grade mechanism of action data is not yet available (DrugBank query pending confirmation). Based on the mechanistic rationale captured in this evidence pack, clofarabine is a second-generation purine (deoxyadenosine) nucleoside analog that inhibits DNA polymerase α/ε and ribonucleotide reductase, depleting the intracellular deoxynucleotide pool and triggering the mitochondrial apoptosis pathway. This gives it direct cytotoxic activity against rapidly dividing cells — a mechanism well suited to hematologic malignancies with high proliferative fractions.

Clofarabine's original approved indication, pediatric relapsed/refractory ALL, and the predicted new indication, myeloid leukemia (AML/CML), are both hematologic malignancies driven by highly proliferative blast populations in the bone marrow. Because the drug's antileukemic mechanism is not lineage-restricted (it targets DNA synthesis machinery common to both lymphoid and myeloid blasts), extensive off-label and investigational use in adult and pediatric AML has already accumulated over two decades, including single-agent Phase II studies and multiple combination regimens (clofarabine + cytarabine, + idarubicin, + busulfan-based transplant conditioning). This existing body of AML-directed research is the mechanistic and clinical bridge supporting the TxGNN prediction.


Clinical Trial Evidence

Trial Number Phase Status Enrollment Key Findings
NCT01423175 Phase 2 Unknown 60 Randomized comparison of ClAraC (clofarabine + Ara-C) vs. FLAMSA conditioning in high-risk AML/advanced MDS prior to allogeneic SCT
NCT01794702 Phase 1/2 Completed 65 Decitabine followed by clofarabine + idarubicin + cytarabine (CIA) in acute leukemia; dose-finding and disease control
NCT00044889 Phase 2 Completed 40 Single-arm, open-label clofarabine monotherapy in adult refractory/relapsed AML
NCT00852709 Phase 1 Terminated 35 Dose-escalation of clofarabine followed by fractionated cyclophosphamide in relapsed/refractory pediatric acute leukemias
NCT00908167 Phase 1 Completed 44 Sorafenib sequenced with cytarabine and clofarabine in relapsed/refractory AML, APL, ALL and infantile leukemia
NCT00454480 Phase 2/3 Completed 2000 Large treatment-development program for older AML/high-risk MDS patients, incorporating clofarabine-containing arms
NCT00932412 Phase 2 Completed 735 Randomized CLARA (clofarabine/intermediate-dose cytarabine) vs. high-dose cytarabine as AML consolidation in younger patients
NCT02665065 Phase 3 Active, not recruiting 153 Iomab-B plus reduced-intensity conditioning (clofarabine-containing regimens common in this population) vs. conventional care in relapsed/refractory AML
NCT00373529 Phase 2 Completed 116 Single-agent clofarabine in previously untreated older AML patients unlikely to benefit from intensive induction
NCT01295307 Phase 2 Completed 86 Clofarabine-based salvage therapy in relapsed/refractory AML as a bridge to allogeneic HCT

Literature Evidence

PMID Year Type Journal Key Findings
31246522 2019 Phase III RCT J Clin Oncol AML08 multicenter randomized trial: clofarabine can replace anthracyclines/etoposide in remission induction for childhood AML
32187883 2020 Phase 2 Cohort Cancer Medicine Clofarabine + cytarabine + mitoxantrone (CLAM) in refractory/relapsed AML: high response rates, effective bridge to allo-HCT
36336258 2023 Cohort Transplant Cell Ther Clofarabine + busulfan myeloablative conditioning for allogeneic HCT in active myeloid malignancies
27621503 2015 Review/Cohort Hospital Pharmacy Practical review of clofarabine + cytarabine regimen preparation and administration for AML
31281098 2019 Review Lancet Oncology Summary of clofarabine and cytarabine combination data in AML
25457773 2015 Review Crit Rev Oncol Hematol Role of clofarabine in adult AML across monotherapy and combination strategies
22957815 2013 Review Leukemia & Lymphoma Clofarabine's mechanism (ribonucleotide reductase/DNA polymerase inhibition) and role in AML
23526416 2013 Review Am J Hematol AML risk-stratification and management update, contextualizing nucleoside-analog therapy
18433953 2008 Review Blood Reviews Conventional and novel treatment approaches for AML in the elderly
17852710 2007 Review Leukemia & Lymphoma "Clofarabine: past, present, and future" — mechanism and development history in leukemia

Finland Market Information

Clofarabine is currently not marketed in Finland (0 marketing authorizations on file). No product listings are available to summarize.


Cytotoxicity

Item Content
Cytotoxicity Classification Conventional cytotoxic (purine nucleoside analog / antimetabolite)
Myelosuppression Risk High — nucleoside analogs that inhibit DNA synthesis are expected to cause profound myelosuppression (neutropenia, thrombocytopenia, anemia); drug-specific incidence figures are pending package-insert confirmation
Emetogenicity Classification Please refer to the package insert warnings and precautions
Monitoring Items CBC with differential, liver and renal function, electrolytes, signs of infection
Handling Protection Must follow institutional cytotoxic drug handling and disposal regulations

Safety Considerations

Please refer to the package insert for safety information. (Key warnings, contraindications, and drug-interaction data are currently unavailable — TFDA package insert retrieval is a blocking data gap for this candidate.)


Conclusion and Next Steps

Decision: Proceed with Guardrails

Rationale: Clofarabine's mechanism of action against high-proliferation blast cells, combined with a substantial body of Phase I–III clinical evidence in AML (including a randomized Phase III pediatric trial and a large randomized Phase II consolidation study), supports an L2 evidence level for the myeloid leukemia indication. However, the drug is not currently marketed in Finland and core safety documentation is missing.

To proceed, the following is needed:

  • TFDA/EMA package insert with warnings, precautions, and contraindications (blocking gap — DG001)
  • Confirmed mechanism of action data from DrugBank (DG002)
  • Formal drug-drug interaction (DDI) review, currently unresolved (query status: not found)
  • Route-of-administration compatibility assessment between required IV administration and any future local formulation
  • Regulatory pathway evaluation given the current "not marketed" status in Finland

    Disclaimer

This content is for research purposes only and does not constitute medical advice. Clinical validation is required before any clinical application.



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